50+ EP
This podcast is all about Real Estate investing and Mortgages, Business and Economic News Updates and Entrepreneurship
249Today
Business
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Discover amazing podcasts created by real people on Jellypod.Curated selection updated daily with fresh content
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50+ EP
The Remnant Awakens is a powerful prophetic truth podcast hosted by Pierre Lidji, a 25-year-old Yahudiy descendant from the tribe of Yahudah, lineage of Shem, Noach 2nd son. This episode series ignites the hearts of the 12 tribes of Yashar’el and grafted-in believers by revealing the true identity of the Hebrews, exposing religious deception, unpacking Scripture with power, and preparing the remnant for the Second Exodus and the return of the King. Stay tuned!
981 week ago
Religion & Spirituality
50+ EP
AI news you can trust—decoded in 10 minutes or less. Co-hosted by Nova Drake and Sam Guss, this sharp, fast-paced show breaks down the top AI stories from around the world three times a week. Each episode blends headline summaries, expert analysis, and behind-the-scenes context—revealing not just what’s happening in AI, but why it matters. Think 60 Minutes for AI—with clarity, edge, and no hype.
781 month ago
Technology
50+ EP
I am Dr. Chelsea McGee AND Welcome to The Real 411—where we make it our mission to dive deeper, think bigger, and challenge the narrative. Every day, we are flooded with headlines. These headlines are quick, catchy, and often misleading. News is everywhere, but how much of it is the complete truth? How often do we stop and ask, "What's really going on?" Because here’s the thing—the story is never just the headline.
641 month ago
News
50+ EP
Modern Service Mastery is Business School with Boots On—built for the people who fix, build, manage, and move the world forward. If you’re in landscaping, remodeling, HVAC, or any hands-on profession, this series was made for you. Each chapter is short, focused, and designed to meet you where you are: behind the wheel, on the jobsite, or planning with your team. You’ll get podcast-style audio, quick written takeaways, and gamified momentum to help you grow as a leader in the real world.
562 months ago
Education
Misc promos for generating with other podcasts in other voices.
492 days ago
Business
Reflections Unfiltered is a podcast exploring the rich, complex experiences of a life lived to the fullest by John Harvey (AKA DIGITALLYWIRED) and friends. From the grit of policing to the nuances of engineering, philosophy, and the occult, I dive into the lessons, insights, and both humorous and sobering realizations along my journey. Using AI, and my own voice we are getting the story out.
471 month ago
Education
The New Sentinel is a podcast dedicated to providing in-depth discussions and deep dives into a wide array of topics. From the glitz and glamour of pop culture to the strategic intricacies of military operations, we cover it all. Each episode is thoughtfully crafted to offer listeners a comprehensive understanding of the subject matter, featuring expert interviews, detailed analysis, and engaging storytelling. Whether you're a pop culture enthusiast or someone with a keen interest in global security, The New Sentinel has something for everyone. Tune in to stay informed and entertained.
461 week ago
News
Join us for real talk about immigration law that makes sense. This podcast breaks down immigration news, policy changes, and visa updates without the legal jargon. Each episode features candid conversations between experienced attorneys. From breaking news to step-by-step visa guidance, we cover what matters most to immigrants, families, and employers. Understanding your options shouldn't require a law degree—tune in for the immigration conversation your community needs. The information contained in this podcast is provided for informational purposes only and should not be construed as legal advice on any subject matter. You should not act or refrain from acting on the basis of any content included in this podcast without seeking legal or other professional advice. The contents of this podcast contain general information and may not reflect current legal developments or address your situation. We disclaim all liability for actions you take or fail to take based on any content on this site. The operation of this site does not create an attorney-client relationship between you and Shankar & Associates, PC.
463 weeks ago
Government
Welcome to the EnableUs Community Podcast! Dedicated to uncovering practical, proven ways to connect with the right NDIS participants. Each concise episode delivers expert insights, compliance essentials, marketing strategies and authentic stories from providers who have successfully grown their client base. Tune in and transform your participant outreach today, fast.
462 months ago
Business
New & Noteworthy
Current events that piss people off
2Today
News
A podcast to help me better digest the information of Concepts 1.
3Yesterday
Education
Step into a time machine with two curious 5th grade teachers as they journey across centuries to meet people from different cultures and eras. Each episode features lively conversations with kids and adults from the past, who share what life was really like in their homes, villages, and communities. From daily routines to cultural traditions, and from special ceremonies to major historical events, listeners will get a front-row seat to history as if they were there. Perfect for classrooms, families, and young history buffs, this podcast makes the past come alive with curiosity, humor, and discovery.
4Yesterday
Education
[Podcast Intro Music] Host: "Welcome, footy fans, to The Final Siren, your go-to AFL podcast for all the action, analysis, and excitement around the AFL Finals! I’m your host, [Name], and today, we’re previewing the 2025 AFL Finals Series! The top 8 is set, and we’ve got some mouth-watering matchups. Whether you're a die-hard supporter or just looking to keep up, we’ve got you covered. So, let’s dive in!" Segment 1: How We Got Here — The Road to Finals Host: "It’s been a massive season, hasn’t it? Some real surprises in the 2025 AFL campaign. Teams like GWS Giants and Gold Coast Suns breaking through to secure finals spots — who would’ve seen that coming? And let’s not forget the usual heavyweights like Brisbane Lions, Collingwood Magpies, and the Geelong Cats, who’ve been solid all year." Co-Host or Guest (Optional): "Absolutely, [Host]. The way the competition has evolved this season is just crazy. Adelaide Crows started strong, but it’s been the rise of teams like Fremantle Dockers that’s really caught my eye. They've found that winning formula and they're heading into the finals with confidence." Host: "Yeah, Fremantle has really turned it on in the second half of the season. And let’s not forget the surprise factor from teams like Hawthorn Hawks — they've been playing some incredible footy lately and could easily cause an upset." Segment 2: The Teams — Who’s In, Who’s Out? Host: "So, let’s break down who’s made it into the 2025 AFL Finals. The top 8 teams this year are: Adelaide Crows Geelong Cats Brisbane Lions Collingwood Magpies GWS Giants Fremantle Dockers Gold Coast Suns Hawthorn Hawks Now, that’s a stacked top 8! Looking at the top 4, I think Brisbane Lions and Collingwood Magpies are looking really strong. Both have been in good form all season. But don’t count out the Geelong Cats, who have that finals experience." Co-Host or Guest: "Definitely! Geelong Cats have been here before, and you can’t underestimate their ability to perform under pressure. And then, of course, Adelaide Crows have been really impressive this season — especially with [Player Name] leading the charge. If they can maintain that intensity, they’ll be tough to beat." Host: "Right, Adelaide has been a real standout team this year. But, you know, I’m also super curious about GWS Giants. They've been under the radar for most of the season, but if they come out firing in the finals, they could shake things up." Segment 3: Key Matchups — Breaking Down the First Week of Finals Host: "Let’s get to the fun part: the first week of finals matchups. We’ve got some cracking games lined up, and here’s how they’re shaping up: Adelaide Crows vs. Gold Coast Suns Geelong Cats vs. Hawthorn Hawks Brisbane Lions vs. Fremantle Dockers Collingwood Magpies vs. GWS Giants The first game between Adelaide Crows and Gold Coast Suns is going to be a real test for the Suns, isn’t it?" Co-Host or Guest: "Absolutely. Adelaide has the advantage of playing at home, and they've got that red-hot form, but don’t sleep on Gold Coast. They've got a youthful energy that could catch Adelaide off guard. The Crows will need to shut down [Player Name] early if they want to stay in control." Host: "Right, Gold Coast has some explosive players. As for the Geelong Cats and Hawthorn Hawks matchup, that one’s going to be a tactical battle. Geelong has the depth, but Hawthorn is unpredictable. You never know what you’re going to get with them in finals." Co-Host or Guest: "I think the Brisbane Lions and Fremantle Dockers game will be an absolute cracker too. Fremantle’s been in top form lately, but the Lions have been dominant for most of the season. That midfield battle is going to be crucial." Segment 4: X-Factors & Players to Watch Host: "Now, we can’t talk finals without talking X-factors. Which players are going to step up and carry their teams when it matters most? For me, I’m looking at [Player Name] from Fremantle Dockers. They’ve been on fire lately, and if they can get their hands on the ball early, they could turn the game in Fremantle’s favour." Co-Host or Guest: "Good shout! I’m going with [Player Name] from Brisbane Lions. This is a guy who can take the game by the scruff of the neck and really dictate the tempo. If he’s on his game, he could break the Dockers’ defence apart." Host: "And don’t forget [Player Name] from Adelaide Crows — they’ve been leading from the front all season and in the finals, you know they’ll rise to the occasion." Segment 5: Predictions — Who’s Going All the Way? Host: "So, we’ve broken down the matchups, talked about the key players, but now, the big question: Who’s going all the way in 2025?" Co-Host or Guest: "I think Brisbane Lions have to be the favourites. They’ve had such a strong season, and with players like [Player Name] and [Player Name] firing, they look well-placed for a deep run. But, I wouldn’t count out Collingwood Magpies — they’ve got the experience and the game plan to go all the way." Host: "I’m leaning towards Geelong Cats. You can never underestimate a team with finals pedigree. Plus, they’ve got some great match-ups across the field that could see them rise above the competition." Segment 6: Listener Questions — Fan Predictions Host: "Alright, let’s see what the fans are predicting. We’ve got a question here from [Listener Name] asking, ‘Which team has the biggest chance of causing an upset in the finals?’" Co-Host or Guest: "Great question! I think GWS Giants could surprise a few people. They’ve been playing some exciting footy, and with a few key players really starting to click, they could cause some headaches for the higher-ranked teams." Host: "Good call. For me, I think Hawthorn Hawks could be the wildcard. They’ve been pretty inconsistent, but if they hit their stride, anything can happen in finals footy." Outro Host: "That’s a wrap for our 2025 AFL Finals Preview! It’s shaping up to be an electrifying series, and we can’t wait to see how it all unfolds. Be sure to subscribe, rate, and review if you love the pod. Drop us your finals predictions in the comments or on social media — we’d love to hear from you! Thanks for tuning in to The Final Siren, and we’ll be back next week after the first week of finals action." [Podcast Outro Music]
22 days ago
Sports
Create an episode where Oliver Bennett teaches beginner learners how to describe their home and different rooms using simple English vocabulary and phrases with practical examples for everyday conversations.
4Yesterday
Education
The Masters weave hidden designs across nations, steering power, belief, and progress. Their aim: absolute dominion through control of minds, resources, and destiny’s unfolding arc.
22 days ago
Society & Culture
Class one of anatomy: Describe functional anatomy within the broader context of occupational therapy theory and practice. Explain foundational concepts of kinesiology, physics, and biomechanics as they apply to purposeful movement and occupational performance. Recognize the relationships between the design and function of anatomical structures of the human body. arthrokinematics axis of motion biomechanics closed-chain movement elasticity end-feel first-class lever force functional anatomy
22 days ago
Education
Las experiencias, enseñanzas y recomendaciones de santos, como Santa Teresa de Ávila para los que comulgan.
22 days ago
SpanishReligion & Spirituality
Cells: Cell Definitions Photosynthesis Structure of the leaf: Waxy cuticle: thin, waterproof layer that reduces water loss Upper Epidermis: Transparent protective layer, allows light to pass through Palisade mesophyll: Packed with chloroplasts Spngy Mesophyll: Allows for gas exchange Vascular bundle: Xylem for water, phloem for nutrients Lower epidermis: Where most stomata are found Include word equation (Water + Carbon Dioxide (sunlight)→ Oxygen + Glucose) Include chemical equations (6CO2 + 6H2O → 6O2 + C6H12O6) Organelles: a specialised, membrane-bound structure within a eukaryotic cell that performs a specific function, analogous to an organ in a larger organism Stroma is transparent: lacks chlorophyll, contains nutrients and water How do plants get their reactants? Water: Through osmosis in the root hair cells Carbon dioxide: Through diffusion in the stomata The process of photosynthesis? LDR: Water is broken down into H+ ions, electrons and oxygen gas. Uses light energy to produce NADPH and ATP in the thylakoid membrane LIR: Uses a series of reactions to bind H+ ions to CO2 to produce glucose using the NADPH and ATP from LDR in the stroma Factors that affect the rate? Less water → less H+ ions, less ATP → less glucose Less CO2 → Less glucose Limiting factors → increasing one will increase the rate TO A POINT where one will become limiting Less sunlight → LDP cannot function High Temperature = faster particles, too high = denature enzyme Enzymes? High temperature → Permanent Denature Low temperature → Slows the rate of reaction Denature → No ATP produced → no glucose produced → photosynthesis stops Adaptations? Clear membrane → Allows for better light absorption in chloroplasts Closer to the edge of the cell for better absorption Thylakoid stacks → Increases surface area for absorption Stroma close to grana → close proximity leads to a faster transfer of products Shade leaves thin → to maximise surface area for absorption they have thinner cuticles and also less palisade cells to minimise water loss Respiration Anaerobic (Glucose → Lactic acid + 2 ATP) Cytoplasm Advantages of Anaerobic? Does not require oxygen Faster Disadvantages of Anaerobic? Less ATP produced Produces toxic byproducts Why can only carry out Anaerobic respiration for short periods of time? Because the production of lactic acid build up causes fatigue and Aerobic (Oxygen + Glucose → Water + Carbon dioxide + 36 ATP) Glycolysis is similar to Anaerobic respiration. Krebs cycle: Mitochondrial matrix ETC: Mitochondrial cristae Advantages of Aerobic? Produces 18x more ATP Does NOT produce toxic byproducts Disadvantages of Aerobic? Slower Requires oxygen Mitochondria number? The higher the mitochondria number in the cells = the more energy the cells use Cell Cycle Checkpoints to ensure that the cell is healthy (not cancerous) Ensures that the DNA is safe to replicate Cell grows DNA replication Mitosis/Meiosis How does the cell cycle prevent mutations? The cell cycle has checkpoints that can ensure that the cell is functioning properly. This allows for the process to stop growing the cell, in order to prevent the cell from possibly being cancerous or harmful to the organism. DNA replication BEFORE Mitosis/Meiosis Purpose: “to produce two identical copies of a cell's DNA, ensuring that each new daughter cell receives a complete and accurate set of genetic instructions during cell division” DNA: Wrapped around histones Double helix Stores genetic information Antiparallel strands Deoxyribose (5-carbon sugar) + Phosphate group + nitrogenous base Leading (5’ to 3’) and lagging strands (3’ to 5’) Helicase unzips the DNA strand New nucleotides bind to the exposed strand from 5’ to 3’ DNA Polymerase uses Complementary base pairing (A to T, C to G) Leading and lagging strands Lagging strand in sections called Okasaki fragments Ligase binds the fragments to make a complete strand Semi-conservative replication (one old and one new strand) Name the enzymes? Helicase, DNA polymerase, ligase Mitosis Prophase: Chromosomes condense and become visible Metaphase: Sister chromatids line up at the cell equator Anaphase: The spindle separates the chromosomes Telophase: The cells begin to split as the cytoplasm divides Cytokinesis: 2 complete identical daughter cells are formed Diffusion Passive Simple diffusion Facilitated Diffusion Osmosis → Hypotonic to hypertonic Hypertonic Hypotonic e.g. root hair cells for osmosis, alveoli for gas exchange, ion pumps in neurons. Active Active transport e.g. Ion pump Factors that affect the rate? Surface Area Concentration Temperature Distance of diffusion Why Active transport can bring in more substances? Because the rate of facilitated diffusion gets slower the closer the concentration of the substance inside and outside the cell is (equilibrium), while active transport can continue to go against the concentration gradient because it uses energy. Enzymes Substrate specific Made up of proteins and hydrogen bonds May require co-factors or co-enzymes Co factors = Inorganic molecules (usually metal ions) Co enzymes = organic molecules. Reduce activation energy Catabolic = Break down Anabolic = Build up Induced fit model Rate of enzymes? Enzyme concentration Substrate concentration Temperature Low temperatures → Slow down High temperature → Denature Presence of inhibitors Competitive → Bind directly to the active site Non-competitive → Binds to the outside, changes active site shape pH (un optimal pH can break the hydrogen bonds) Denature = Lose its function Genetic Variation Meiosis Purpose → to produce gametes with half the chromosome number, restoring diploidy at fertilisation. Fertilisation itself also creates variation (random fusion of gametes). Prophase 1: Chromosomes condense and become visible Crossing over - new combination of ALLELES (not genes) Metaphase 1: Homologous chromosomes line up at at cell equator Independent assortment - each gamete only receives one chromosome Anaphase 1: The spindle separates homologous chromosomes Telophase 1: The cells begin to split as the cytoplasm divides Metaphase 2: Chromosomes line up at the equator Anaphase 2: Chromosomes are pulled apart by spindle fibers Segregation Telophase 2: The cells begin to split as the cytoplasm divides Cytokinesis: 4 unique daughter cells are formed Where is variation? Crossing over → increases the chance of recombination, resulting in different phenotypes from the parents Independent assortment → Mixes the mothers' and fathers' genes Segregation → mixes the mothers' and fathers' alleles Fertilisation → combination of 2 unique gametes Genes and alleles Homozygous Dominant, Homozygous Ressesive, Heterozygous Different ways of dominance? Co-dominance - Both alleles FULLY expressed. Ratio: 1:2:1 Incomplete dominance - Blend of both alleles. Ratio: 1:2:1 Complete dominance - Only the dominant allele is expressed. Ratio 3:1 Explain the differences between genotype and phenotype ratios? Because the incomplete/co-dominace results is a blend and splotch expression of the alleles, leading to 3 different phenotypes rather than the 2 possible phenotypes from the 3 genotypes in complete dominance. Linked genes: genes found on the same chromosome Why does independent assortment not affect linked genes? Unless crossing over has occurred, the genes are on the same chromosome and cannot be separated. Why does crossing over not affect non-linked genes? Because the genes are already on each chromosome, they cannot be crossed over. Sex-linked genes: genes only found on X or Y chromosomes (x linked is more common in males) Why is it more common for males to inherit it compared to females? Because males only have 1 X chromosomes, and females have 2, meaning if it is recessive, the female needs to have 2 copies of the allele while the male still only needs one. Multiple alles: When there is more than 2 alleles for a single gene Lethal alleles: Alleles that significantly reduce the lifespan of the organism that posses them. Ratio 2:1 Charts: Pedigree: Looking at a specific family for inheritance Monohybrid inheritance: inheritance of 1 gene (2 alleles) Dihybrid inheritance: inheritance of 2 genes (4 alleles) Effects on Gene pool Allele frequency Fixed alleles: 0% or 100% frequency Founders effects Bottleneck effect Genetic drift Mutations (original and only source of new alleles) Natural selection Population size: Smaller population more susceptible to changes Gene flow How are new alleles introduced and spread in a population? Mutations in the base sequence is the only new source of alleles. Natural selection selects advantageous alleles by those organisms that posses those advantageous alleles living long enough to reproduce, spreading their alleles to their offspring. This increases the allele frequency in the gene pool. Beneficial alleles increase in allele frequencies, harmful alleles decrease in allele frequency Explain Founder's effect, genetic drift, bottleneck effect and natural selection Gene Expression Genetic variation: the naturally occurring differences in alleles (versions of genes) and genetic information within a population or species The process by which the information in a gene is used to synthesise the other product. Transcription Promoter region, coding region, terminator region RNA polymerase matches the nucleotides to the template (anti-sense) strand in the complementary base pairing rule of A-U, C-G. Not the coding (sense) strand. Editing phase Introns (non coding parts of mRNA) are spliced out and the exons (coding parts of mRNA) are joined together Translation When mRNA goes through the ribosome, and the tRNA is able to match the codon on the mRNA to complementary anticodon on the tRNA, until it reaches a stop codon, to ensure the correct amino acid sequence Point mutations: Substitution → No change in reading frame Deletion, Insertion → Big change in reading frame, which can alter the protein, removing its intended function. Same-sense: No new amino acid is formed Mis-sense: A new amino acid is formed Non-sense: No new amino acid is formed Protein structure: Primary: Polypeptide chain, peptide bonds Secondary: Alpha jelix or beta sheets, hydrogen bonds Tertiary: 3D structure, di-sulphide bridges, hydrogen bonds, ionic bonds Quantenary: Combination of tertiary structures Types of proteins Enzymatic, structural, regulatory, transport Metabolic pathways: Enzyme-catalysed reactions where the product of one of the reactions is the reactant of another reaction. When an excess of one product is produced, it can inhibit a previous reaction intentionally to regulate, or unintentionally due to faulty gene. Cycle metabolic pathways always produce the starting reactant as the final product How can 2 parents have a metabolic mutation but offspring does not? As long as the offspring inherits one functioning allele, it is ables to go through the full metabolic pathway. Mutagens: environmental factor that causes a mutation eg carcinogens (cancer causing) Mutations are PERMANENT Epigenetic markers can control the expression of genes (whether they are transcribed or not, based on methyl and acetyl groups). These can be controlled through external or internal factors that do not change the genotype. Cline is a gradual change in the phenotype over an environmental gradient Type of RNA Structure Function Stage mRNA Short, unstable, single-stranded RNA, corresponding to a gene encoded within DNA Serves as intermediary between DNA and protein; used by ribosome to direct synthesis of protein it encodes Both tRNA Short (70-90 nucleotides), stable RNA with extensive intramolecular base pairing; contains an amino acid binding site and an mRNA binding site Carries the correct amino acid to the site of protein synthesis in the ribosome Translation rRNA Longer, stable RNA molecules composing 60% of ribosome’s mass Ensures the proper alignment of mRNA, tRNA, and ribosome during protein synthesis; catalyzes peptide bond formation between amino acids Translation Why is mRNA unstable while the others are stable? mRNA is temporar and easy to degrade (only lived for purpose and cna be controlled to save energy) tRNA and rRNA is structurally protected and reused many times (saves cell energy from remaking them) What are 3 similarities between DNA and RNA? Both are Nucleic Acids - made of nucleotides, sugars, phosphates and bases Both are used in the cell to undergo protein synthesis - DNA for genetic information and RNA for translation of DNA triplets to RNA transfer and translate it into proteins Both use complementary base pairing to create new strands - A-T or A-U and C-G What are 3+ differences between DNA and RNA? DNA cannot leave the nucleus, and RNA can - because DNA needs to be kept safe inside the nucleus DNA uses Thymine and RNA uses Uracil - 2 different bases that bind to Adenine DNA is double-stranded, while RNA is single-stranded DNA uses Deoxyribose and RNA uses Ribose How can mutations in DNA result in faulty metabolic pathway? When a mutation in the DNA occurs, the proteins synthesis of that gene can turn the gene into a protein that forms an enzyme. If there was any mutation that resulted in a different protein forming, the function of the enzyme would not be able to be achieved, therefore, the reactants cannont be converted into the products if there is not correct gene/allele for the enzyme. The product would not be produced. The organism needs atleast one functioning allele to code for the correct enzyme, in order for the enzyme to function. What happens if a premature stop codon is produced? A premature stop codon causes translation to end too early, producing a shortened (truncated) protein. Because it is incomplete, it cannot fold into the correct shape, so it loses its function. This loss of function is significant because it can be harmful to the organism. What would happen if a stop codon was removed? If a stop codon is removed, the ribosome keeps adding amino acids until it reaches another stop codon. If this is close, the protein may still function, but usually the extra amino acids cause misfolding. This prevents the protein from functioning correctly and can be harmful to the organism. Effect of non-mutagen vs mutagen? Non-mutagens can trigger epigenetic markers to be turned on or off, resulting in a different expression of the phenotype without changing the genotype. Mutagens change the genotype and therefore the phenotype changes. How triplets, codons and anticodons work together? Triplets in the template strand (3 sequential nucleotides on a DNA strand) code for the codons in the mRNA. When the RNA polymerase adds the free nucleotides onto the template strand it produces codons (3 sequential nucleotides on a mRNA strand that code for amino-acids). When the mRNA goes in the translation phase the cytoplasm, the tRNA has anticodons (complementary to the codons on the mRNA strand) to ensure that the aminoacids on the tRNA are in the correct order. The chain of the amino acids produce proteins. 2 or more reasons why DNA cannot be directly transcribed into a polypeptide chain? The DNA needs to be protected in the nucleus from possible mutations that may occur in exposure in the cytoplasm and in the translation process. The tRNA that carries the amino acids is specifically shaped to be complementary to the mRNA strand. There needs to be multiple proteins produced at the same time from the same DNA strand which would not be possible as each cell only has 1 pair of chromosomes.
23 days ago
Education
Every song hides a story. In the Life Lyrics Podcast, Rachel sits down with Paul — the songwriter behind Life Lyrics — to uncover the meaning behind his most intimate lyrics. From Paris cafés to wedding vows whispered at the altar, each episode explores the emotions, stories, and hidden moments that inspired the music. Together, they guide listeners into an emotional sanctuary where love, hope, heartbreak, and honesty live side by side. Whether you’re here for the songs or the stories, you’ll leave feeling a little more human, and maybe… a little more seen.
11Yesterday
Music
International Podcasts
Black Box Society taucht ein in die verborgensten Geheimnisse unserer Zeit – von Tech-Verschwörungen über geheime Experimente bis hin zu verborgenen Netzwerken der Macht. Jede Episode verbindet Fakten mit spekulativen Erzählungen, erschaffen durch modernste Algorithmen, die Realität und Fiktion verschmelzen lassen. Was ist wahr, was nur eine perfekte Simulation? Finde es heraus – oder verliere dich in den Mustern der Information.
225 months ago
GermanSociety & Culture
Denne podcast tale som hvilke forudsætninger virksomhederne har for rent faktisk at opnå succes.
184 days ago
DanishBusiness
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